研究文章
TNRC6C函数作为一个肿瘤抑制和经常在乳头状甲状腺癌表达下调
图3
TNRC6C调节增殖、凋亡、迁移和入侵BCPAP细胞的能力。(a)与TNRC6C-siRNA1 BCPAP细胞转染,TNRC6C-siRNA2, TNRC6C-siRNA3和control-SiRNA分别。相对的mRNA表达TNRC6C被实时qPCR量化。(b)与pcDNA3.1-TNRC6C BCPAP细胞转染质粒和pcDNA3.1-empty质粒,分别。相对的mRNA表达TNRC6C被实时qPCR量化。(c) BCPAP细胞的生长曲线由CCK8检测转染后TNRC6C-SiRNA3或control-SiRNA。(d) BCPAP细胞的生长曲线由CCK8化验与pcDNA3.1-TNRC6C质粒转染后或pcDNA3.1-empty质粒。(e)的影响TNRC6C击倒或超表达BCPAP细胞的迁移使用愈合试验评估。定量分析(左)和代表图像(右)。(f)的影响TNRC6C击倒或超表达BCPAP由流式细胞术测定细胞凋亡。 (g) The effect of TNRC6C knockdown or overexpression on the migration of BCPAP cells was assessed using transwell migration assay. Quantitative analysis (left) and representative images (right). (h) The effect of TNRC6C knockdown or overexpression on the invasion of BCPAP cells was assessed using transwell invasion assays (quantitative analysis (left) and representative images (right). Values represent the mean ± SD from three independent experiments; empty-plasmid, pcDNA3.1-empty plasmid; TNRC6C-plasmid, pcDNA3.1-TNRC6C plasmid;
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