文摘
的分泌肿瘤坏死factor-α(TNFα)interleukin-1α(IL-α)和白细胞介素- 6 (il - 6)研究了人类星形细胞瘤细胞的罚款1 h, 3 h,感染后6 h和24小时从三个刚地弓形虫菌株(RH毒性;cystogentc, 76 k和Prugniaud菌株)。星形细胞瘤细胞分泌TNFα起来从而il - 6,但没有IL-1α。积极的控制是通过刺激与佛波醇酯诱导显著增加(
) in TNFα and IL- 6 secretion but not in IL-1α, while lipopolysaccharide (alone and after priming), interferon gamma, ionophore A 23187 and sera positive to T. gondii did not induce any increase in cytokine levels. None of the tachyzoites, whatever their virulence, induced a significant increase in cytokine production at any time in the study. Tachyzoites did not inhibit the secretion induced by phorbol esters.">
的分泌肿瘤坏死factor-α(TNFα)interleukin-1α(IL-α)和白细胞介素- 6 (il - 6)研究了人类星形细胞瘤细胞的罚款1 h, 3 h,感染后6 h和24小时从三个刚地弓形虫菌株(RH毒性;cystogentc, 76 k和Prugniaud菌株)。星形细胞瘤细胞分泌TNFα起来从而il - 6,但没有IL-1α。积极的控制是通过刺激与佛波醇酯诱导显著增加(