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在反义理解:非编码rna在Diabetes-Induced血管功能障碍的治疗潜力
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microrna参与血管病理生理学。ECs:内皮细胞;VSMCs:血管平滑肌细胞;SIRT1:沉默交配类型信息监管2同族体;ITGA5:整合素亚单位5;JAK1: Janus激酶1;KLF: Kruppel-like因素;ELK1: ETS ELK1 domain-containing蛋白质;PPAR:过氧物酶体proliferator-activated受体;SPROUTY2: sprouty蛋白2;SEMA6A: semaphorin-6A;SPRED1: sprouty-related EVH1 domain-containing蛋白1;GATA2: GATA-binding蛋白2;p21 PAK4: protein-activated激酶4;PI3KR2: phosphatidylinositide 3-kinase调节亚基2;VCAM1:血管细胞粘附分子1;低氧诱导因子:低氧诱导因子1;ZEB1:锌指E-Box绑定同源框1; PTEN: phosphatase and tensin homolog; PDCD4: programmed cell death 4; CAMK2D: calcium/calmodulin-dependent protein kinase II delta; PDGF: platelet-derived growth factor; ACE: angiotensin-converting enzyme; KIT: v-kit Hardy-Zuckerman 4 feline sarcoma viral oncogene homolog; CDKN1B: cyclin-dependent kinase inhibitor 1B.
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